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NSW CTP Claim
NSW CTP

Peripheral nerve injury

Lower-limb peripheral nerve injury after a motor accident

A peripheral nerve injury is different from a spinal nerve-root or spinal cord injury. The CTP assessment identifies the named nerve, objective motor and sensory deficit and any dysaesthetic pain, then applies the Part 6 lower-limb nerve method without duplicating strength, gait, atrophy or CRPS.

Lower-limb motor, sensory, reflex and electrodiagnostic evidence reviewed for NSW CTP.
Peripheral nerve WPI requires anatomical localisation and objective deficit, not tingling or radiating pain alone.

Motor accident injury

How can this injury happen?

Car or passenger collision

Pelvic, femoral, knee or fibular trauma can stretch, compress or divide the sciatic, peroneal or tibial nerve.

Motorcycle accident

A motorcycle crush or traction injury may damage a named lower-limb nerve.

Pedestrian or cyclist impact

Impact or fracture around the pelvis, knee or leg can injure a nerve directly or through compartment pressure.

Injuries that can occur

  • sciatic, common peroneal or tibial nerve injury
  • femoral, obturator or other named peripheral nerve lesion
  • traction, compression, laceration or post-surgical nerve injury
  • motor loss, sensory loss or dysaesthetic pain
  • foot drop or focal muscle denervation

Symptoms and functional problems

  • foot drop, weakness or altered walking
  • numbness in an anatomical distribution
  • burning, electric or touch-evoked dysaesthetic pain
  • reduced balance or difficulty stairs and pedals

Seek urgent medical assessment

New progressive weakness, foot drop, saddle symptoms, bladder/bowel change or threatened circulation requires urgent assessment for central, root or limb pathology.

Clinical evidence

What findings matter?

The diagnosis should localise the lesion and distinguish peripheral nerve injury from radiculopathy or central nervous system injury. Examination, anatomy, EMG/NCS and associated fracture or surgical records should agree.

Record or examinationWhat it may establishWhat it cannot prove alone
Motor and sensory examinationMaps muscle power and reproducible sensory deficit to a named nerve.General weakness or tingling does not establish the nerve or severity.
EMG and nerve-conduction studiesMay support lesion site, axonal loss and chronic denervation.A test result must match the clinical distribution and causation.
Imaging and operative recordsShows fracture, compression, laceration or decompression/repair.Imaging proximity alone does not prove functional nerve loss.

Movement in daily life

How movement affects real activities

Motor loss may affect ankle dorsiflexion, plantar flexion, toe control or hip/knee function. The movement consequence is described clinically, but the same nerve loss is not rated again under strength, gait or atrophy.

Dorsiflexion and toe extension

Foot clearance and avoiding trips.

Common peroneal motor deficit is assessed through the named-nerve method.

Plantar flexion and intrinsic foot control

Push-off, balance and prolonged walking.

Tibial motor/sensory deficit is anatomically assessed.

Sensation and dysaesthesia

Protective feeling, footwear tolerance and contact sensitivity.

Sensory deficit and dysaesthetic pain are addressed within the permitted nerve method.

Threshold injury is a separate question: verified nerve injury may be non-threshold. Paraesthesia or radiating pain without objective nerve injury may remain threshold.

Part 6 permanent impairment

How is CTP WPI assessed?

Clauses 6.104 to 6.106 use AMA4 section 3.2k and Table 68, with motor, sensory and dysaesthetic components combined. Guidelines Table 6.3 supplies selected maximum nerve values and the result is converted through Table 6.4.

Measurement rules that apply

  • Clauses 6.69 and 6.70 require the method that most specifically addresses the lower-limb impairment. Gait should not replace a joint, nerve, fracture or replacement method that can be applied reliably.
  • Clause 6.84 requires active range of motion, a goniometer where clinically indicated and consistent repetitions when reliability is uncertain. Passive movement may inform the examination but does not set the impairment value.
  • Clause 6.85 says only the most severe deficit in one direction or axis from the same lower-limb ROM table is rated. Deficits from separate tables may be combined only as the Guidelines permit.
MethodCTP sourceWhen it is relevantImportant limit
Named peripheral nerve deficitClauses 6.104-6.106; AMA4 Table 68Objective motor, sensory and dysaesthetic deficit in a named nerve.Cannot combine with strength, gait, atrophy or CRPS.
Selected maximum valuesGuidelines Table 6.3Sciatic: motor 30% WPI, sensory 7%, dysaesthesia 5%; common peroneal: 15%, 2%, 2%; tibial: 15%, 5%, 3%.These are maximum values, not automatic ratings.
Lower-limb conversionClause 6.110; Guidelines Table 6.4Conversion where the method produces lower-extremity impairment.Do not add another method for the same deficit.
  • Localise the named peripheral nerve.
  • Grade motor, sensory and dysaesthetic components using the permitted method.
  • Do not use gait, strength, atrophy or CRPS for the same nerve injury.

What cannot be combined?

  • peripheral nerve with lower-limb strength
  • peripheral nerve with gait or muscle atrophy
  • peripheral nerve with CRPS for the same injury

What does not establish WPI by itself?

  • radiating pain
  • tingling without reproducible loss
  • EMG abnormality without clinical correlation
  • foot drop without anatomical diagnosis

Motor accident examples

Fibular-head trauma with common peroneal palsy

The distribution, power, sensation and EMG may support a named-nerve assessment; Table 6.3 values remain maxima, not automatic awards.

Leg pain from lumbar radiculopathy

That is assessed as a spinal nerve-root issue, not automatically as a lower-limb peripheral nerve injury.

Claim file preparation

Evidence checklist

neurologist or rehabilitation diagnosis naming the nerve
serial MRC motor and mapped sensory findings
EMG/NCS raw findings and interpretation
fracture, operative or decompression records
orthosis use and functional rehabilitation evidence
dated GP, hospital and specialist records describing the accident mechanism and first lower-limb findings
weight-bearing status, walking aids, gait and active joint measurements recorded over time
prior imaging and records for the same limb where causation or deduction is in issue
rehabilitation, capacity and work-task evidence showing the practical residual impairment

Assessment source

Lower-limb nerve WPI source

Assessment source: Motor Accident Guidelines v10.1 clauses 6.104-6.110; AMA4 section 3.2k and Table 68; Guidelines Tables 6.3 and 6.4.

Threshold injury: Verified peripheral nerve injury may be non-threshold; symptoms without objective nerve injury may remain threshold.

What the assessor checks

  • named-nerve method
  • motor/sensory/dysaesthetic components
  • selected maximum values
  • prohibited combinations

What does not establish the result by itself

  • pain
  • tingling
  • EMG alone
  • weakness without localisation

Official sources

Related NSW CTP guides

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Frequently asked questions

Is radiating leg pain a peripheral nerve injury?
No. It may reflect a spinal root, peripheral nerve or another cause and requires anatomical findings.
Does an EMG result set WPI?
No. It supports localisation and severity but must match the clinical deficit.
Are Table 6.3 figures automatic?
No. They are maximum values for selected nerves and components.
Can gait or strength be added?
No. Clause 6.106 prohibits combining the nerve method with strength, gait or atrophy.
Can CRPS also be rated?
Not for the same lower-limb nerve injury; clause 6.106 prohibits that combination.